2nd International Conference on

Pharmaceutical Chemistry

Paris | France   July 16-17, 2018

Ian Martins

Ian Martins

Edith Cowan University School of Medicine


Dr Martins completed his undergraduate Science degree at the University of Western Australia in 1984, then completing a MSc in Clinical Biochemistry in 1986. In 1987 he commenced a Ph D studying the role of high density lipoprotein (HDL) in diabetes (Hackett Scholarship). In 1993 he was awarded a Saw fellowship from the University of Western Australia to conduct studies relating to the metabolism of chylomicrons and their remnants and the influence of size and composition on their clearance from blood and its importance to atherosclerosis. Dr. Ian Martins is a Reveiwer for Open Acess Pub and various other international journals. Appointed as the Chief Editor for International Journal of Diabetes Research (2014-2017) and an editor for Research and Reviews: Neuroscience (2016-2017) and various other journals. He is a BIT Member (BIT Congress. Inc) with an H-index of 43, (ResearchGate STATs (23), Mendeley STATS (20). He is now a Scientist for The Science Advisory Board (USA) and an Academic with Academia.edu. Data analysis and interpretations indicate that global organ disease by various biomarker tests may not diagnose programmed cell death with relevance to severity of disease progression. The total citations over the past 27 years of scientific research has accumulated to 3000.

Research Interest

His current research into links between diet, diabetes and neurodegenerative diseases builds upon his past experiences and generates outputs that will have relevant and significant impact to the community. The incidence of diabetes has been predicted to increase to 21% by 2050. In various continents the rise in the global diabetes epidemic has been associated with diseases of various organ diseases related to obesity, diabetes and neurodegenerative diseases (Parkinson's disease and Alzheimer's disease). Novel and critical elements of scholarly peer review provides information that the heat shock gene Sirtuin 1 is repressed with relevance to diabetes, neurological diseases and Alzheimer's disease. Major unsolved challenges to advances in biology may now provide evidence to reverse Sirtuin 1 dysfunction that is connected to primarily to heat shock protein metabolism and secondly to toxic amyloid beta metabolism with mitochondrial apoptosis in liver and brain cells. In the developing world increased levels of bacterial lipopolysaccharides repress the anti-aging gene Sirtuin 1 with the development of diabetes, neurodegenerative diseases and Alzheimer’s disease. Unhealthy diets accelerate Type 3 diabetes with the induction of NAFLD in Global Populations.

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